Search results for " Glutamate"

showing 10 items of 97 documents

Involvement of the glutamate receptor AtGLR3.3 in plant defense signaling and resistance toHyaloperonospora arabidopsidis

2013

Like their animal counterparts, plant glutamate receptor-like (GLR) homologs are intimately associated with Ca(2+) influx through plasma membrane and participate in various physiological processes. In pathogen-associated molecular patterns (PAMP)-/elicitor-mediated resistance, Ca(2+) fluxes are necessary for activating downstream signaling events related to plant defense. In this study, oligogalacturonides (OGs), which are endogenous elicitors derived from cell wall degradation, were used to investigate the role of Arabidopsis GLRs in defense signaling. Pharmacological investigations indicated that GLRs are partly involved in free cytosolic [Ca(2+)] ([Ca(2+)]cyt) variations, nitric oxide (N…

0106 biological sciencesArabidopsis thaliana[SDV]Life Sciences [q-bio]ArabidopsisOligosaccharidesPlant Science01 natural sciencesCALCIUM SIGNATURESchemistry.chemical_compoundGene Expression Regulation PlantSYSTEMIC ACQUIRED-RESISTANCEArabidopsisPlant defense against herbivoryArabidopsis thalianaPlant ImmunityGENE-EXPRESSIONCalcium signaling0303 health sciencesIMMUNE-RESPONSESTOBACCO CELLSfood and beveragesCYTOSOLIC CALCIUMElicitorOomycetesReceptors GlutamateBiochemistryHost-Pathogen Interactions[SDE]Environmental SciencesoligogalacturonidesSignal transductionSignal Transductionglutamate receptorHyaloperonospora arabidopsidisBiologyNitric Oxidecalcium signaling03 medical and health sciencesplant defenseGeneticsDNQX[SDV.BV]Life Sciences [q-bio]/Vegetal BiologyBOTRYTIS-CINEREA030304 developmental biologyHyaloperonospora arabidopsidisNITRIC-OXIDEArabidopsis ProteinsCell Biologybiology.organism_classificationSALICYLIC-ACIDchemistryPLASMA-MEMBRANEReactive Oxygen Species010606 plant biology & botanyThe Plant Journal
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Involvement of putative glutamate receptors in plant defence signaling and NO production

2011

International audience; Ionotropic glutamate receptors (iGluRs) are non-selective cation channels permeable to calcium, present in animals and plants. In mammals, glutamate is a well-known neurotransmitter and recently has been recognized as an immunomodulator. As animals and plants share common mechanisms that govern innate immunity with calcium playing a key role in plant defence activation, we have checked the involvement of putative iGluRs in plant defence signaling. Using tobacco cells, we first provide evidence supporting the activity of iGluRs as calcium channels and their involvement in NO production as reported in animals. Thereafter, iGluRs were shown to be activated in response t…

0106 biological sciencesHypersensitive responsebiochemistry and molecular biologyplant defenceglutamate receptorCell Culture TechniquesGlutamic AcidBiologycalcium signaling01 natural sciencesBiochemistrytobaccoFungal Proteins03 medical and health sciencesnitric oxideelicitorsExcitatory Amino Acid Agonists[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biologyrésistance végétalePlant Proteins030304 developmental biologyCalcium signaling0303 health sciencesVoltage-dependent calcium channelAlgal ProteinsGlutamate receptorGeneral MedicineGlutamic acidImmunity InnateElicitortabacReceptors GlutamateBiochemistryMetabotropic glutamate receptorNMDA receptorCalciumExcitatory Amino Acid Antagonists010606 plant biology & botany
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Regulation of Dendritic Spine Morphology in Hippocampal Neurons by Copine-6.

2015

Dendritic spines compartmentalize information in the brain, and their morphological characteristics are thought to underly synaptic plasticity. Here we identify copine-6 as a novel modulator of dendritic spine morphology. We found that brain-derived neurotrophic factor (BDNF) - a molecule essential for long-term potentiation of synaptic strength - upregulated and recruited copine-6 to dendritic spines in hippocampal neurons. Overexpression of copine-6 increased mushroom spine number and decreased filopodia number, while copine-6 knockdown had the opposite effect and dramatically increased the number of filopodia, which lacked PSD95. Functionally, manipulation of post-synaptic copine-6 level…

0301 basic medicineDendritic spineVesicular Inhibitory Amino Acid Transport Proteinsdrug effects [Synapses]Tropomyosin receptor kinase BHippocampal formationgenetics [Carrier Proteins]pharmacology [Brain-Derived Neurotrophic Factor]Hippocampusmetabolism [Vesicular Inhibitory Amino Acid Transport Proteins]Mtap2 protein ratMice0302 clinical medicineNeurotrophic factorsdrug effects [Synaptic Vesicles]genetics [Nerve Tissue Proteins]Cells Culturedultrastructure [Neurons]NeuronsChemistryLong-term potentiationSynaptic Potentialsphysiology [Neurons]physiology [Dendritic Spines]Cell biologyultrastructure [Dendritic Spines]metabolism [Receptor trkB]Synaptic VesiclesFilopodiaultrastructure [Synaptosomes]Disks Large Homolog 4 ProteinMicrotubule-Associated ProteinsCognitive NeuroscienceDendritic Spinesmetabolism [Disks Large Homolog 4 Protein]Nerve Tissue Proteinsgenetics [Receptor trkB]03 medical and health sciencesCellular and Molecular NeuroscienceOrgan Culture Techniquesphysiology [Synaptic Vesicles]metabolism [Vesicular Glutamate Transport Protein 1]TrkB protein ratdrug effects [Synaptic Potentials]Synaptic vesicle recyclingAnimalsHumansReceptor trkBddc:610metabolism [Synaptosomes]metabolism [Nerve Tissue Proteins]Viaat protein ratBrain-Derived Neurotrophic Factormetabolism [Microtubule-Associated Proteins]Rats030104 developmental biologygenetics [Synaptic Potentials]nervous systemcytology [Hippocampus]Synaptic plasticityultrastructure [Synapses]SynapsesVesicular Glutamate Transport Protein 1CPNE6 protein ratphysiology [Synapses]Carrier Proteins030217 neurology & neurosurgerymetabolism [Carrier Proteins]SynaptosomesCerebral cortex (New York, N.Y. : 1991)
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Oral Monosodium Glutamate Administration Causes Early Onset of Alzheimer's Disease-Like Pathophysiology in APP/PS1 Mice.

2019

Glutamate excitotoxicity has long been related to Alzheimer's disease (AD) pathophysiology, and it has been shown to affect the major AD-related hallmarks, amyloid-β peptide (Aβ) accumulation and tau phosphorylation (p-tau). We investigated whether oral administration of monosodium glutamate (MSG) has effects in a murine model of AD, the double transgenic mice APP/PS1. We found that AD pathogenic factors appear earlier in APP/PS1 when supplemented with MSG, while wildtype mice were essentially not affected. Aβ and p-tau levels were increased in the hippocampus in young APP/PS1 animals upon MSG administration. This was correlated with increased Cdk5-p25 levels. Furthermore, in these mice, we…

0301 basic medicineGenetically modified mouseMalemedicine.medical_specialtyMonosodium glutamateExcitotoxicityHippocampusAdministration OralMice TransgenicAMPA receptormedicine.disease_cause03 medical and health scienceschemistry.chemical_compoundAmyloid beta-Protein PrecursorMice0302 clinical medicineOral administrationAlzheimer DiseaseInternal medicinemental disordersSodium GlutamatemedicinePresenilin-1Animalsbusiness.industryGeneral NeuroscienceGlutamate receptorLong-term potentiationGeneral MedicineFlavoring AgentsPsychiatry and Mental healthClinical Psychology030104 developmental biologyEndocrinologychemistryFemaleGeriatrics and Gerontologybusiness030217 neurology & neurosurgeryJournal of Alzheimer's disease : JAD
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Piriform cortex alterations in the Ts65Dn model for down syndrome

2020

The piriform cortex is involved in olfactory information processing, that is altered in Down Syndrome. Moreover, piriform cortex has a crucial involvement in epilepsy generation and is one of the first regions affected in Alzheimer's Disease, both maladies being prevalent among Down Syndrome individuals. In this work, we studied the alterations in neuronal morphology, synaptology and structural plasticity in the piriform cortex of the Ts65Dn mouse model, which is the most used model for the study of this syndrome and mimics some of their alterations. We have observed that Ts65Dn piriform cortex displays: a reduction in dendritic arborisation, a higher density of inhibitory synapses (GAD67),…

0301 basic medicineGlutamate decarboxylasePresynaptic TerminalsMice TransgenicPiriform CortexInhibitory postsynaptic potentialMice03 medical and health sciences0302 clinical medicineAtrophyPostsynaptic potentialPiriform cortexmedicineNeuropilAnimalsMolecular BiologyNeuronsGephyrinbiologyGlutamate DecarboxylaseGeneral NeuroscienceMembrane Proteinsmedicine.disease030104 developmental biologymedicine.anatomical_structurenervous systemVesicular Glutamate Transport Protein 1biology.proteinExcitatory postsynaptic potentialNeurology (clinical)Down SyndromeNeuroscience030217 neurology & neurosurgeryDevelopmental BiologyBrain Research
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Differential role of interleukin-1β in neuroinflammation-induced impairment of spatial and nonspatial memory in hyperammonemic rats.

2019

Activated microglia and increased brain IL-1β play a main role in cognitive impairment in much pathology. We studied the role of IL-1β in neuroinflammation-induced impairment of the following different types of learning and memory: novel object recognition (NOR), novel object location (NOL), spatial learning, reference memory (RM), and working memory (WM). All these processes are impaired in hyperammonemic rats. We assessed which of these types of learning and memory are restored by blocking the IL-1 receptor in vivo in hyperammonemic rats and the possible mechanisms involved. Blocking the IL-1 receptor reversed microglial activation in the hippocampus, perirhinal cortex, and prefrontal cor…

0301 basic medicineMaleInterleukin-1betaPostrhinal cortexHippocampusBiochemistryHippocampusReceptors N-Methyl-D-Aspartate03 medical and health sciences0302 clinical medicineReceptors GABAAmmoniaMemoryPerirhinal cortexGeneticsmedicineMemory impairmentAnimalsHyperammonemiaReceptors AMPARats WistarPrefrontal cortexMolecular BiologyNeuroinflammationCerebral CortexInflammationMicrogliabusiness.industryWorking memoryAnimal FeedRatsInterleukin 1 Receptor Antagonist ProteinProtein Subunits030104 developmental biologymedicine.anatomical_structureGene Expression RegulationReceptors GlutamateMicrogliabusinessNeuroscience030217 neurology & neurosurgeryBiotechnologyFASEB journal : official publication of the Federation of American Societies for Experimental Biology
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The Effects of N-Acetylcysteine on the Rat Mesocorticolimbic Pathway: Role of mGluR5 Receptors and Interaction with Ethanol

2021

N-acetylcysteine (NAC) is a prodrug that is marketed as a mucolytic agent and used for the treatment of acetaminophen overdose. Over the last few decades, evidence has been gathered that suggests the potential use of NAC as a new pharmacotherapy for alcohol use disorder (AUD), although its mechanism of action is already being debated. In this paper, we set out to assess both the potential involvement of the glutamate metabotropic receptors (mGluR) in the possible dual effect of NAC administered at two different doses and NAC’s effect on ethanol-induced activation. To this aim, 30 or 120 mg/kg of NAC was intraperitoneally administered to rats with the presence or absence of the negative allo…

0301 basic medicinePharmaceutical ScienceglutamatePharmacologyAcetylcysteine03 medical and health sciencesPharmacy and materia medica0302 clinical medicineNeurochemicalDrug Discoverymental disordersmedicinealcoholismMetabotropic glutamate receptor 5ChemistryCommunicationRGlutamate receptor<i>N</i>-acetylcysteineN-acetylcysteineRS1-441030104 developmental biologyMetabotropic receptorMTEPMechanism of actionMetabotropic glutamate receptorAlcoholismeMedicineMolecular Medicinemedicine.symptomTecnologia farmacèuticaMedicaments030217 neurology & neurosurgerymedicine.drugPharmaceuticals
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Homer2 and alcohol: A mutual interaction

2017

The past two decades of data derived from addicted individuals and preclinical animal models of addiction implicate a role for the excitatory glutamatergic transmission within the mesolimbic structures in alcoholism. The cellular localization of the glutamatergic receptor subtypes, as well as their signaling efficiency and function, are highly dependent upon discrete functional constituents of the postsynaptic density, including the Homer family of scaffolding proteins. The consequences of repeated alcohol administration on the expression of the Homer family proteins demonstrate a crucial and active role, particularly for the expression of Homer2 isoform, in regulating alcohol-induced behav…

0301 basic medicineScaffold proteinlcsh:RC435-571media_common.quotation_subjectMini ReviewAddiction; Alcohol; Glutamate; Homer proteins; Homer2; Psychiatry and Mental HealthglutamateBiologyNucleus accumbensHomer203 medical and health sciencesGlutamatergic0302 clinical medicineExtended amygdalalcsh:PsychiatryNeuroplasticityCellular localizationmedia_commonPsychiatryHomer proteinalcoholAddictionHomer proteins030104 developmental biologyPsychiatry and Mental HealthaddictionNeurosciencePostsynaptic density030217 neurology & neurosurgery
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Changes in Serine Racemase-Dependent Modulation of NMDA Receptor: Impact on Physiological and Pathological Brain Aging

2018

International audience; The N-methyl-D-Aspartate glutamate receptors (NMDARs) are pivotal for the functional and morphological plasticity that are required in neuronal networks for efficient brain activities and notably for cognitive-related abilities. Because NMDARs are heterogeneous in subunit composition and associated with multiple functional regulatory sites, their efficacy is under the tonic influence of numerous allosteric modulations, whose dysfunction generally represents the first step generating pathological states. Among the enzymatic candidates, serine racemase (SR) has recently gathered an increasing interest considering that it tightly regulates the production of D-serine, an…

0301 basic medicine[SDV]Life Sciences [q-bio]Allosteric regulation[SHS.PSY]Humanities and Social Sciences/PsychologyglutamateDiseaseReviewBiologyBiochemistry Genetics and Molecular Biology (miscellaneous)BiochemistryNMDA receptors[SHS.PSY] Humanities and Social Sciences/Psychology03 medical and health sciences0302 clinical medicineserine racemasemedicineMolecular BiosciencesAmyotrophic lateral sclerosislong term potentiationMolecular BiologyPathologicallcsh:QH301-705.5ComputingMilieux_MISCELLANEOUS[SCCO.NEUR]Cognitive science/Neuroscience[SCCO.NEUR] Cognitive science/NeuroscienceagingGlutamate receptorLong-term potentiationAlzheimer's diseasemedicine.diseaseMESH: NMDA receptors serine racemase aging Alzheimer’s disease D-serine long term potentiation glutamate[SDV] Life Sciences [q-bio]030104 developmental biologylcsh:Biology (General)d-serineSerine racemaseNMDA receptor[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Neuroscience030217 neurology & neurosurgeryFrontiers in Molecular Biosciences
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Endocannabinoid LTD in Accumbal D1 Neurons Mediates Reward-Seeking Behavior

2020

Summary The nucleus accumbens (NAc) plays a key role in drug-related behavior and natural reward learning. Synaptic plasticity in dopamine D1 and D2 receptor medium spiny neurons (MSNs) of the NAc and the endogenous cannabinoid (eCB) system have been implicated in reward seeking. However, the precise molecular and physiological basis of reward-seeking behavior remains unknown. We found that the specific deletion of metabotropic glutamate receptor 5 (mGluR5) in D1-expressing MSNs (D1miRmGluR5 mice) abolishes eCB-mediated long-term depression (LTD) and prevents the expression of drug (cocaine and ethanol), natural reward (saccharin), and brain-stimulation-seeking behavior. In vivo enhancement…

0301 basic medicineglutamate02 engineering and technologyMolecular neuroscienceBiologyNucleus accumbensMGLUR5 receptorsMedium spiny neuronArticleinduced reinstatementBehavioral Neuroscience03 medical and health sciencesDopamineDopamine receptor D2lipasemedicinelong-term depression[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]lcsh:ScienceLong-term depressionrelapseMultidisciplinaryMetabotropic glutamate receptor 5021001 nanoscience & nanotechnologyEndocannabinoid systemin-vivo exposure3. Good healthrats030104 developmental biologynervous systemethanol-seekingplasticitylcsh:Q[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Molecular Neuroscience0210 nano-technologyNeurosciencepsychological phenomena and processesNeurosciencemedicine.drugiScience
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